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ERK活化於SCA17小鼠病理機制之意義
Hsieh, Hsiu-Mei
(PI)
Department of Life Science
Project
:
Government Ministry
›
Ministry of Science and Technology
Overview
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INIS
mice
100%
proteins
66%
cerebellum
50%
kinases
33%
apoptosis
33%
motors
16%
phenotype
16%
genes
16%
patients
16%
mitogens
16%
losses
16%
transgenic mice
16%
correlations
16%
expansion
16%
signals
16%
nervous system diseases
16%
aggregation
16%
humans
16%
Neuroscience
Spinocerebellar Ataxia
100%
Gliosis
40%
Cerebellum
30%
TATA-binding Protein
30%
Purkinje Cell
30%
Neuroapoptosis
20%
Immunohistochemistry
10%
Microglia
10%
Astrocyte
10%
Neurodegenerative Disorder
10%
Extracellular Signal-Regulated Kinases
10%
CAG Repeat
10%
Caspase 3
10%
Calbindin
10%
Glia
10%
Behavior (Neuroscience)
10%
Mitogen-Activated Protein Kinase
10%
Cell Loss
10%
Cell Degeneration
10%
Western Blot
10%